mouse monoclonal wisp2 (Santa Cruz Biotechnology)
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Mouse Monoclonal Wisp2, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 21 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/wisp2/WISP-2+Antibody/us12465618-282-21-28
Average 93 stars, based on 21 article reviews
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Incubation:Article Title: Single-Cell Transcriptomics Reveals the Difference of Aortic Atherosclerosis Response to Phytosterols and Oxidation Products of Sterols. Article Snippet: Scope: Phytosterols (PS) and sterol oxidation products are key dietary factors influencing atherosclerosis besides cholesterol, although the mechanisms remain elusive.. Recently, single-cell RNA sequencing (scRNA-seq) has revealed the heterogeneity of multiple cell types associated with complex pathogenesis in atherosclerosis development.. Methods and results: Here, scRNA-seq is performed to investigate the alterations in the aortic cells from ApoE−/− mice induced by diet-derived PS or two sterol oxidation products, phytosterols oxidation products (POPs), and cholesterol oxidation products (COPs). Article Title: Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling. Article Snippet: Accepted Manuscript Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling Xiao-fang Han, Xin-xiu Zhang, Ke-mei Liu, Qiu Zhang PII: S0016-6480(18)30161-8 DOI: https://doi.org/10.1016/j.ygcen.2018.05.024 Reference: YGCEN 12950 To appear in: General and Comparative Endocrinology Received Date: 17 March 2018 Revised Date: 14 May 2018 Accepted Date: 22 May 2018 Please cite this article as: Han, X-f., Zhang, X-x., Liu, K-m., Zhang, Q., Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling, General and Comparative Endocrinology (2018), doi: https://doi.org/10.1016/j.ygcen.2018.05.024 This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript.. The manuscript will undergo copyediting, typesetting, and review of the resulting proof before it is published in its final form. Article Title: A Novel Mechanism of bta-miR-210 in Bovine Early Intramuscular Adipogenesis Article Snippet: .. The membranes were blocked with 5% ( w/v ) nonfat dried milk at room temperature for one hour, and then incubated the target proteins with following primary antibodies against ZNF423 (sc-10486, Santa Cruz Biotechnology, Santa Cruz, CA, USA), PPARγ (sc-6284, Santa Cruz Biotechnology, Santa Cruz, CA, USA), FABP4 (sc-18661, Santa Cruz Biotechnology, Santa Cruz, CA, USA), other:Article Title: Up-regulation of T-cell factor-4 isoform-responsive target genes in hepatocellular carcinoma Article Snippet: Enzyme-linked immunosorbent assay Direct binding duplex enzyme linked immunosorbent assay (ELISA) was performed as previously described ( 17 ). Blocking Assay:Article Title: Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling. Article Snippet: Accepted Manuscript Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling Xiao-fang Han, Xin-xiu Zhang, Ke-mei Liu, Qiu Zhang PII: S0016-6480(18)30161-8 DOI: https://doi.org/10.1016/j.ygcen.2018.05.024 Reference: YGCEN 12950 To appear in: General and Comparative Endocrinology Received Date: 17 March 2018 Revised Date: 14 May 2018 Accepted Date: 22 May 2018 Please cite this article as: Han, X-f., Zhang, X-x., Liu, K-m., Zhang, Q., Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling, General and Comparative Endocrinology (2018), doi: https://doi.org/10.1016/j.ygcen.2018.05.024 This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript.. The manuscript will undergo copyediting, typesetting, and review of the resulting proof before it is published in its final form. Nonspecific Binding:Article Title: Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling. Article Snippet: Accepted Manuscript Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling Xiao-fang Han, Xin-xiu Zhang, Ke-mei Liu, Qiu Zhang PII: S0016-6480(18)30161-8 DOI: https://doi.org/10.1016/j.ygcen.2018.05.024 Reference: YGCEN 12950 To appear in: General and Comparative Endocrinology Received Date: 17 March 2018 Revised Date: 14 May 2018 Accepted Date: 22 May 2018 Please cite this article as: Han, X-f., Zhang, X-x., Liu, K-m., Zhang, Q., Apelin-13 deficiency alters cortical bone geometry, organic bone matrix, and inhibits Wnt/β-catenin signaling, General and Comparative Endocrinology (2018), doi: https://doi.org/10.1016/j.ygcen.2018.05.024 This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript.. The manuscript will undergo copyediting, typesetting, and review of the resulting proof before it is published in its final form. Cell-Signaling:Article Title: Loss of WISP2/CCN5 in Estrogen-Dependent MCF7 Human Breast Cancer Cells Promotes a Stem-Like Cell Phenotype Article Snippet: .. The membranes were blocked with saturating buffer for 1 h at room temperature (25°C), then probed with the following specific antibodies: CD44, actin, LEF1, FOXC1, ZEB1, Claudin-3, Claudin-4, Claudin-7, E-cadherin, IGFBP7, Oct3/4, Nanog and Formalin-fixed Paraffin-Embedded:Article Title: Wnt Signaling Pathway Proteins in Scar, Hypertrophic Scar, and Keloid: Evidence for a Continuum? Article Snippet: Hypertrophic scars and keloids are fibroproliferative lesions characterized by excessive collagen deposition.. It is unclear whether these entities represent distinct disorders or share a common pathogenesis and the molecular underpinnings of these lesions are poorly understood.. Accumulating evidence suggests that the Wnt signaling pathway is a key regulator of wound healing. Immunohistochemical staining:Article Title: Wnt Signaling Pathway Proteins in Scar, Hypertrophic Scar, and Keloid: Evidence for a Continuum? Article Snippet: Hypertrophic scars and keloids are fibroproliferative lesions characterized by excessive collagen deposition.. It is unclear whether these entities represent distinct disorders or share a common pathogenesis and the molecular underpinnings of these lesions are poorly understood.. Accumulating evidence suggests that the Wnt signaling pathway is a key regulator of wound healing. |

